Tag Archives: poison

Research Suggests EPA Standard for Pesticide Safety Overlooks Poisons’ Long-term Effects

We received this press release from the good people at the University of Pittsburgh news section. I think it’s a wake-up call to government agencies charged with regulating pesticides. This gross oversight on the part of the EPA should scare you and make you ask yourself: ‘who is safe these days?’

The dangers that pesticides pose to wildlife is immense and although the researchers in this report used only amphibians, we can all imagine what implication these poisons would have on large mammals and other species. I am particularly reminded of the danger already posed by Furadan on lions and other predators, birds of prey and scavengers. Maybe you need to read along and see this for yourself, for these poisons are not only a danger to wildlife, but also to humans.

August 12, 2009
FOR IMMEDIATE RELEASE
Contact:   Morgan Kelly
[412-624-4356 (office); 412-897-1400 (cell); mekelly@pitt.edu]

Pitt Research Suggests EPA Standard for Pesticide Safety Overlooks Poisons’
Long-term Effects

Environmental Toxicology and Chemistry article reports “lag effect,”
revealing that harmful effects can remain hidden until after EPA’s four-day
direct exposure test

PITTSBURGH-The four-day testing period the U.S. Environmental Protection
Agency (EPA) commonly uses to determine safe levels of pesticide exposure
for humans and animals could fail to account for the toxins’ long-term
effects, University of Pittsburgh researchers report in the September
edition of Environmental Toxicology and Chemistry.

The team found that the highly toxic pesticide endosulfan-a neurotoxin
banned in several nations but still used extensively in U.S. agriculture-can
exhibit a “lag effect” with the fallout from exposure not surfacing until
after direct contact has ended. Lead author Devin Jones, a recent Pitt
biological sciences graduate, conducted the experiment under Rick Relyea, an
associate professor of biological sciences in Pitt’s School of Arts and
Sciences, with collaboration from Pitt post-doctoral researcher John
Hammond. The paper is available on Pitt’s Web site at
http://www.pitt.edu/news2009/Endosulfan.pdf

The team exposed nine species of frog and toad tadpoles to endosulfan levels
“expected and found in nature” for the EPA’s required four-day period, then
moved the tadpoles to clean water for an additional four days, Jones
reported. Although endosulfan was ultimately toxic to all species, three
species of tadpole showed no significant sensitivity to the chemical until
after they were transferred to fresh water. Within four days of being moved,
up to 97 percent of leopard frog tadpoles perished along with up to 50
percent of spring peeper and American toad tadpoles.

Of most concern, explained Relyea, is that tadpoles and other amphibians are
famously sensitive to pollutants and considered an environmental indicator
species. The EPA does not require testing on amphibians to determine
pesticide safety, but Relyea previously found that endosulfan is 1,000-times
more lethal to amphibians than other pesticides. Yet, he said, if the
powerful insecticide cannot kill one the world’s most susceptible species in
four days, then the four-day test period may not adequately gauge the
long-term effects on larger, less-sensitive species.

“When a pesticide’s toxic effect takes more than four days to appear, it
raises serious concerns about making regulatory decisions based on standard
four-day tests for any organism,” Relyea said. “For most pesticides, we
assume that animals will die during the period of exposure, but we do not
expect substantial death after the exposure has ended. Even if EPA
regulations required testing on amphibians, our research demonstrates that
the standard four-day toxicity test would have dramatically underestimated
the lethal impact of endosulfan on even this notably sensitive species.”

Andrew Blaustein, a professor in Oregon State University’s nationally ranked
Department of Zoology, who is familiar with the Pitt study, said the results
raise concerns about standards for other chemicals and the delayed dangers
that might be overlooked. Some of the frog eggs the Pitt team used had been
collected by Blaustein’s students for an earlier unrelated experiment, but
he had no direct role in the current research.

“The results are somewhat alarming because standards for assessing the
impacts of contaminants are usually based on short-term studies that may be
insufficient in revealing the true impact,” Blaustein said. “The
implications of this study go beyond a single pesticide and its effect on
amphibians. Many other animals and humans may indeed be affected similarly.”

Tadpoles in the Pitt project spent four days in 0.5 liters of water
containing endosulfan concentrations of 2, 6, 7, 35, 60, and 296
parts-per-billion (ppb), levels consistent with those found in nature. The
team cites estimates from Australia-where endosulfan is widely used-that the
pesticide can reach 700 ppb when sprayed as close as 10 meters from the
ponds amphibians typically call home and 4 ppb when sprayed within 200
meters. The EPA estimates that surface drinking water can have chronic
endosulfan levels of 0.5 to 1.5 ppb and acute concentrations of 4.5 to 23.9 ppb.

Leopard frogs, spring peepers, and American toads fared well during the
experiment’s first four days, but once they were in clean water, the death
rate spiked for animals previously exposed to 35 and 60 ppb. Although the
other six species did not experience the lag effect, the initial doses of
endosulfan were still devastating at very low concentrations. Grey and
Pacific tree frogs, Western toads, and Cascades frogs began dying in large
numbers from doses as low as 7 ppb, while the same amount killed all green
frog and bullfrog tadpoles.

The endosulfan findings build on a 10-year effort by Relyea to understand
the potential links between the global decline in amphibians, routine
pesticide use, and the possible threat to humans in the future.

A second paper by Relyea and Jones also in the current Environmental
Toxicology and Chemistry expands on one of Relyea’s most notable
investigations, a series of findings published in Ecological Applications in
2005 indicating that the popular weed-killer Roundup® is “extremely lethal”
to amphibians in concentrations found in the environment. The latest work
determined the toxicity of Roundup Original Max for a wider group of larval
amphibians, including nine frog and toad species and four salamander
species. The report is available on Pitt’s Web site at
http://www.pitt.edu/news2009/Roundup.pdf

In November 2008, Relyea reported in Oecologia that the world’s 10 most
popular pesticides-which have been detected in nature-combine to create
“cocktails of contaminants” that can destroy amphibian populations, even if
the concentration of each individual chemical is within levels considered
safe to humans and animals. The mixture killed 99 percent of leopard frog
tadpoles and endosulfan alone killed 84 percent.

A month earlier, Relyea published a paper in Ecological Applications
reporting that gradual amounts of malathion-the most popular insecticide in
the United States-too small to directly kill developing leopard frog
tadpoles instead sparked a biological chain reaction that deprived them of
their primary food source. As a result, nearly half the tadpoles in the
experiment did not reach maturity and would have died in nature.

News releases about Relyea’s previous work are available on Pitt’s Web site
at http://www.news.pitt.edu

###

8/12/09/tmw

Pictures of poisoned lions vultures in Mara

Dear Friends

I hope you won’t be offended when I show these photos which are so shocking and sad that they will make you cry, then demand an immediate response from FMC and the Kenya Government for hesitating over the ban on Furadan and carbofuran.

Poisoned lion cub Masai mara 25 May 2009

Dead lion cub estimated to be only 8 months old. He was in a pride of 6 that fed on the poisoned carcass.  Nobody knows what happened to the others.

dead lion stomach contents - Masai Mara

Stomach contents of dead lion cub contained parts of the cow (this is it’s tongue) that was laced with a pinkish poison suspected to be Furadan – a carbofuran based pesticide that was widely available in Kenya until the recent buy back by FMC. It is still available in some stores.

Poisoned vultures in Masai Mara

36 vultures of several species are known to have died in this poisoning incident. Others may have flown of and died elsewhere.

some of the 36 vultures killed in Mara

Two people were responsible for this act, and according to KWS one has been arrested, the other fled across the border into Tanzania.
“County council rangers revealed that prior to poisoning; lions had attacked and killed four (4) cattle from larger herd of cattle that were grazing in the reserve at night. The owners of the cattle were seen slaughtering and transporting the meat of the killed cattle on a bicycle. It was therefore suspected that they carried all the meat and finally poisoned one of the bovine carcasses intentionally to kill lions and other wildlife which had attacked their herd. It was their way of retaliating for the loss of their cattle”.

carcas laced with poison in Masai Mara

In conclusion KWS state that

“This is the second time when the lions have suffered from poisoning in Mara, in April, 2008, a pride of 6 male lion got poisoned from yet unidentified source near Mara Serena lodge along Mara river, two of the lions died at the scene while the rest disappeared with clinical signs of paralysis and incoordination gait and were presumed dead. This was likely to be an incidental poisoning after the lions fed on a hippo carcass that apparently died after grazing on a vegetable farm sprayed with a pesticide, due to indirect exposure and less concentration of the chemical on the hippo carcass, the severity of the symptoms in lions was mild and death only occurred after 2 – 3 days.

The poisoning this month was acute and very severe, presented with an instant death soon after feeding on the carcass. This was an intentional poisoning as opposed to the previous one, and involved deaths of several vultures already confirmed and examined. Previous poisoning was confirmed at the Government chemist as carbamates (Carbofuran) which is sold in Kenya as Furadan.

It is very likely that the same Carbofuran (Furadan) chemical has been used to poison animals again this time. The laboratory results will confirm this”.